Gastric Ulcers


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Gastric ulcers are so common in racing horses that many equine practitioners maintain their racing patients on anti-ulcer medications to prevent and treat gastric ulcers. Reports in the literature place the percentage of racing horses in training with endoscopically visible gastric ulcers at grater than 80%.[1] Gastric ulcers are also associated with considerable morbidity related to chronic epigastric pain, nausea, vomiting, and anemia. [2] Gastric ulcers are a round or oval and run perpendicular to the gastric wall. Frequently mucosal folds radiate away from the crater in a "spoke-like" fashion.

Gastric ulcers are a frequent problem in the United States. Proton pump inhibitors have been shown to increase healing rates and improve clinical symptoms.[4] Gastric ulcers are a common problem in athletic horses. Gastric ulcers can result in abdominal pain, but can also result in poor performance.[5] Gastric ulcers are more common in people over the age of 50.As with acute stress gastritis, stress ulcers can occur under the stress of severe illness, skin burns, or trauma. Stress ulcers occur in the stomach and the duodenum.[6]

Gastric ulcers are most common in males between the ages of 55 and 70. [7] Gastric ulcers are less common, with about 85,000 new cases diagnosed each year. They most often affect patients 55-65 years of age.[8]

Gastric ulcers are common in young chinchillas and are frequently caused by feeding coarse, fibrous roughage or moldy feeds. Clinically affected animals may be anorectic or asymptomatic.[9]

Gastric ulcers are more common in people over the age of 60. Men have more problems with duodenal ulcers than women.[10] Gastric ulcers are deep, nonhealing mucosal defects in the stomach, usually in the antrum. Duodenal ulcers are nonhealing defects in the mucosal lining of the duodenum, usually in the duodenal bulb.[11] Gastric ulcers are most often localized on the lesser curvature of the stomach. The ulcer is a round to oval parietal defect ("hole"), 2 to 4 cm diameter, with a smooth base and perpendicular borders.[12]

Gastric ulcers are more common in people aged over 50. [13] Gastric ulcers are generally treated more aggressively because they can take longer to heal. However, there are other ways to support the healing of your ulcer naturally.[14]

Gastric ulcers are generally caused because there is an imbalance between pepsin (an enzyme), stomach acid, and the normal protection of the lining of the stomach. Such an imbalance results in inflammation and burning pain.[15]

Gastric ulcers are a different matter altogether however, as you may know. After the cure for ulcers was discovered, > irritable bowel syndrome (IBS) became the leading reason people went to > gastroenterologists.[16] Gastric ulcers are peptic ulcers in the stomach. Duodenal ulcers are peptic ulcers in the intestine.[17]

Stomach or gastric ulcers which are in the stomach are the second most common and most often affect middle aged or elderly women. [18] Stomach ulcers usually produce mild recurrent bouts of colic and do not usually lead to death. However, sometime these ulcers can perforate the stomach lining and lead to severe peritonitis and death.[19] Stomach distress can be caused by a variety of conditions, including indigestion, dyspepsia, heartburn, bloating, and gastritis - and ulcers. Precise diagnosis is often difficult, because symptoms can be deceptive.[20]

Stomach acid also protects you by killing fungi, bacteria, and viruses that you cannot help ingesting along with your food. Without gastric acid’s protection, you may be much more susceptible to food poisoning, parasites, and other gastrointestinal afflictions — including ulcers.[21]

Gastrinomas occur with equal frequency among men and women, yet duodenal ulcers with hyperacid secretion not due to gastrinomas occur almost exclusively in men, UAB studies report. An ongoing, 17-year prospective study examined more than 80 acid hypersecreting patients (including 60 with Zollinger-Ellison syndrome) taking individually optimized doses of lansoprazole. Gastrin, in turn, stimulates the production of gastric acid by parietal cells. The acid erodes the mucosa and causes the ulcer.